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《Medical Recapitulate》 2015-02
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Progress in Alteration of Bone Metabolism in Corticosteroid-Induced Osteonecrosis of Femoral Head

XU Hai-tao;SHENG Jia-gen;Department of Orthopedics,Shanghai Jiaotong University Affiliated Sixth People's Hospital;  
The clinical treatment of glucocorticoid is a significant risk factor of non-trauma osteonecrosis of femoral head( ONFH). Long-term exogenous glucocorticoids( GC) exposure can affect the proliferation and differentiation of bone marrow stromal cells and the balance between osteoblasts and osteoclasts,which impacts the bone metabolisms and leads to ONFH. GC can inhibit the remodeling of bone and cause osteoporosis,which increases the risk of collapse of femoral head. It is important to prevent ONFH when treating patients with GC. ONFH may be prevented by appropriate drug therapy. The study of the alteration of bone metabolism in GC-induced ONFH may provide reference for the clinical treatment.
【CateGory Index】: R681.8
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1 ZHANG Chun-lei1,ZHOU Sen-ye1,WANG Li-ming2.(1.Department of Orthopedics,Shuyang Hospital of TCM,Shuyang 223600,China;2.Department of Orthopedics,the First Hospital of Nanjing,Affiliated to Nanjing Medical University,Nanjing 210006,China);The Progress in Diagnosis and Treatment of Adult ONFH[J];医学综述;2011-13
【Co-citations】
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1 XU Hai-tao;SHENG Jia-gen;Department of Orthopedics,Shanghai Jiaotong University Affiliated Sixth People's Hospital;;Progress in Alteration of Bone Metabolism in Corticosteroid-Induced Osteonecrosis of Femoral Head[J];医学综述;2015-02
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